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The earliest computers used magnetic tape to store data This is still done in some systems You can get a tape drive for making an emergency backup of the data on your hard drive, or for archiving data you rarely need to use Magnetic tape has very high storage capacity There are microcassettes that can hold more than 1 GB of data; standard cassettes can hold many gigabytes But tapes are extremely slow because, unlike their disk-shaped counterparts, they are a serial-access storage medium This means that the data bits are written in a string, one after another, along the entire length of the tape The drive might have to mechanically rewind or fast-forward through a football field s length of tape to get to a particular data bit, whereas on a disk medium, the read/write head never has to travel further than the diameter of the disk to reach a given data bit

Despite the marked reduction in the medical use of barbiturates during the past three decades, the improper use of these drugs particularly their nonmedical and illicit use is still responsible for a sizeable number of suicides, accidental deaths, and addiction In the past, about 50 barbiturates were marketed for clinical use, but only a few are now encountered with any regularity: pentobarbital (Nembutal), secobarbital (Seconal), amobarbital (Amytal), thiopental (Pentothal), and phenobarbital (Luminal) The rst three are the ones most commonly abused Barbiturates are also a component of combination preparations for the treatment of migraine (eg, butalbital in Fiorinal)

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Severe intoxication occurs with the ingestion of 10 to 20 times the oral hypnotic dose The patient cannot be roused by any means, ie, is comatose Respiration is slow and shallow or irregular, and pulmonary edema and cyanosis may be present The tendon re exes are usually but not invariably absent Most patients show no response to plantar stimulation, but in those who do, the responses are extensor With deep coma, the corneal and gag re exes may also be abolished Ordinarily the pupillary light re ex is retained in severe intoxication and is lost only if the patient is asphyxiated; but in advanced cases, the pupils become miotic and poorly reactive, simulating opiate intoxication At this point respiration is greatly depressed and oculocephalic and oculovestibular re ex responses are usually abolished In the early hours of coma, there may be a phase of exor or extensor posturing or rigidity of the limbs, hyperactive re exes, ankle clonus, and extensor plantar signs; persistence of these signs indicates that anoxic damage has been added The temperature may be subnormal, the pulse is thready and rapid, and the blood pressure is at shock levels Failure of respiration to quicken on painful stimulation is an ominous sign There are few conditions other than barbiturate intoxication that cause a accid coma with small reactive pupils, hypothermia, and hypotension A pontine hemorrhage may do so, but a hysterical trance or catatonic stupor do not present a problem in differential diagnosis The use of gas and high-pressure liquid chromatography provides a reliable means of identifying the type and amount of barbiturate in the blood A patient who has also ingested alcohol may be comatose with relatively low blood barbiturate concentrations Contrariwise, the barbiturate addict may show only mild signs of intoxication with very high blood barbiturate concentrations In mild intoxication, the normal electroencephalogram (EEG) activity is replaced by fast (beta) activity, in the range of 20 to 30 cycles per second, and is most prominent in the frontal regions In more severe intoxication, the fast waves become less regular and interspersed with 3- to 4-per-second slow activity; in still more advanced cases, there are short periods of suppression of all activity, separated by bursts of slow (delta) waves of variable frequency In extreme overdosage, all electrical activity ceases This is one instance in which a at (isoelectric) EEG cannot be equated with brain death, and the effects are fully reversible unless anoxic damage has supervened Management In mild or moderate intoxication, recovery is the rule and special treatment is not required except to prevent aspiration If the patient is unresponsive, special measures must be taken to maintain respiration and prevent infection An endotracheal tube should be inserted, with suctioning as necessary Any risk of respiratory depression or underventilation requires the use of a positive-pressure respirator Hemodialysis or hemo ltration with charcoal may be used in comatose patients who have ingested long-acting barbiturates and these treatments are particularly advisable if anuria or uremia has developed Treatment with analeptic drugs (eg, picrotoxin, pentylenetetrazol), which enjoyed a brief period of popularity, has been generally abandoned, since these drugs do not affect the rate of metabolism or the excretion of barbiturates Occasionally, in the case of a barbiturate addict who has taken an overdose of the drug, recovery from coma is followed by the development of abstinence symptoms, as described later Barbiturate Addiction (Chronic Barbiturate Intoxication) Chronic barbiturate intoxication, like other drug addictions, tends to develop on a background of some psychiatric disorder, most.

In a computer, the term random-access memory (RAM) refers to integrated circuits (ICs) that store working data The amount, and speed, of memory is a crucial factor in determining what a computer can and cannot do

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